your ignorance does not surprise me..
Let's first define Albinism though:
Albinism is the PARTIAL or TOTAL loss of pigmentation. It affects the color of the skin, the color of the hair and the color of the eyes and it can affect the texture of the hair.
What this means is that there are people/Whites who are still albino WITH pigmentation. This is OCA2.
ANY LOSS OF PIGMENTATION IS ALBINISM. Albinism can also cause straightening of the hair
Good, now we've got that out of the way.
There are 4 types of Albinism. Each type affects the pigmentation of the hair, skin and eye color.
This information is NOT hard to find. I got this from the NATIONAL INSTITUTE of HEALTH: AKA NIH
Here is the link, it was done in 2004, it has NOW been found that the mutation of the alanine allele (SCL24A5-an African gene for pigmentation) accounts for the THR111 gene found in Whites and THIS IS WHY they are more genetically related to Asians.
Oculocutaneous albinism - Genetics Home Reference
THR111 Alleles
http://paspcr.med.umn.edu/Commentary...commentary.pdf
Allelic Variations of SLC24A5
There are many genes that affect skin pigmentation, what it doesn't take away is the fact that ANY LOSS IN PIGMENTATION IN THE HAIR, EYES AND SKIN, no matter how minute is in fact ALBINISM.
It's not meant to be mean, it's just a fact. I always though there was just one type!
Oculocutaneous Albinism Type 1
Disease characteristics. Oculocutaneous albinism type 1 (OCA1) is characterized by reduced synthesis of melanin in the skin, hair, and eyes, associated with ocular findings of nystagmus, reduced iris pigment with iris translucency, reduced retinal pigment, foveal hypoplasia with significantly reduced visual acuity usually in the range of 20/100 to 20/400, and misrouting of the optic nerves resulting in alternating strabismus and reduced stereoscopic vision. Individuals with OCA1A have white hair, white skin that does not tan, and fully translucent irises that do not darken with age. At birth, individuals with OCA1B have white or very light yellow hair that darkens with age, white skin that over time develops some generalized pigment and may tan with sun exposure, and blue irises that change to green/hazel or brown/tan with age. Visual acuity may be 20/60 or better in some individuals.
Oculocutaneous albinism, type 2
Tyrosinase-positive oculocutaneous albinism (OCA, type II) is an autosomal recessive disorder in which the biosynthesis of melanin pigment is reduced in skin, hair, and eyes. Although affected infants may appear at birth to have OCA type I, or complete absence of melanin pigment, most patients with OCA type II acquire small amounts of pigment with age. Individuals with OCA type II have the characteristic visual anomalies associated with albinism, including decreased acuity and nystagmus, which are usually less severe than in OCA type I. OCA type II is the most common type of oculocutaneous albinism in the world.
The gene OCA2, when in a variant form, the gene causes the pink eye color and hypopigmentation common in human albinism. Different SNPs within OCA2 are strongly associated with blue and green eyes. Hair color is the pigmentation of hair follicles due to two types of melanin, eumelanin and pheomelanin. Generally, if more melanin is present, the color of the hair is darker; if less melanin is present, the hair is lighter. Blond hair can have almost any proportion of phaeomelanin and eumelanin, but both only in small amounts. More phaeomelanin creates a more golden blond color, and more eumelanin creates an ash blond. Blond hair is common in many European peoples, but rare among peoples of non-European origin. Many children born with blond hair develop darker hair as they age. Red hair ranges from vivid strawberry shades to deep auburn and burgundy, and is the rarest fully distinct hair color on earth. It is caused by a variation in the Mc1r gene and believed to be recessive. Red hair has the highest amounts of phaeomelanin and usually low levels of eumelanin, and is the rarest natural human hair color.
Oculocutaneous albinism, type 3
Oculocutaneous albinism, type 3: A rare inherited disorder characterized by slightly reduced pigmentation in the skin, eyes and hair (due to a genetic mutation of the TYRP1 gene). Type 3 is characterized by some pigmentation of the iris despite the complete absence of tyrosinase which is needed for the production of melanin which gives the skin, hair and eyes their color. Those with OCA3 may have, Reddish-brown skin, Ginger hair, Red hair, Hazel iris, Brown iris. Type 3 also has the milder eye problems than the other types.
Light-skinned Whites with tyrosinase-negative albinism have pale skin and hair color ranging from white to yellow; their pupils appear red because of translucent irides. Blacks with the same disorder have hair that may be white, faintly tinged with yellow, or yellow-brown. Both Whites and Blacks with tyrosinase-positive albinism grow darker as they age. For instance, their hair may become straw-colored or light brown and their skin cream-colored or pink. People with tyrosinase-positive albinism may also have freckles and pigmented nevi that may require excision.
This form of albinism was referred to as 'rufous oculocutaneous albinism (ROCA)' when it was found in southern African blacks. In blacks the disorder is characterized by bright copper-red coloration of the skin and hair and dilution of the color of the iris. Manga et al. (1997) suggested that albinism caused by mutation in the TYRP1 gene should be referred to as OCA3
In New York City rather numerous cases are seen in Puerto Rican families from the Aguadilla-Arecibo area of northwestern Puerto Rico. Albinism in dark-skinned persons such as Puerto Ricans is not always obvious because freckled skin and reddish hair may be present. Red reflex on transillumination of the iris and nystagmus are important clues to the diagnosis.
In tyrosinase-variable albinism, at birth the child's hair is white, his skin is pink, and his eyes are gray. As he grows older, though, his hair becomes yellow, his irides may become darker, and his skin may even tan slightly. The skin of a person with albinism is easily damaged by the sun. It may look weather-beaten and is highly susceptible to precancerous and cancerous growths. The patient may also have photophobia, myopia, strabismus, and congenital horizontal nystagmus.
Oculocutaneous Albinism, Type 4
Disease characteristics. Oculocutaneous albinism type 4 (OCA4) is characterized by hypopigmentation of the skin and hair plus the characteristic ocular changes found in all other types of albinism, including nystagmus; reduced iris pigment with iris translucency; reduced retinal pigment with visualization of the choroidal blood vessels on ophthalmoscopic examination; foveal hypoplasia associated with reduction in visual acuity; and misrouting of the optic nerves at the chiasm associated with alternating strabismus, reduced stereoscopic vision, and an altered visual evoked potential (VEP). Individuals with OCA4 are usually recognized within the first year of life because of hypopigmentation of the hair and skin and the ocular features of nystagmus and strabismus. Vision is likely to be stable after early childhood. The amount of cutaneous pigmentation in OCA4 ranges from minimal to near normal. Newborns with OCA4 usually have some pigment in their hair, with color ranging from silvery white to light yellow. Hair color may darken with time, but does not vary significantly from childhood to adulthood. This form of albinism is rarer than OCA2, except in the Japanese population.
22. Squamous-cell carcinoma in situ in a patient with oculocutaneous albinism
Now, of course Caucs are more genetically related to asians. But they they share a common ancestor. the Africans that were over there became you guessed it, ALBINO and that is a genetic mutation causing a difference in the allele genes. Over time they became a sub-species of the originals, all of whom were African. There's a reason it traced back to Africans. I will provide you with ancient pics of Asian statues.
It's funny how Asians (specifically in China, Japan etc...) and Whites are Caucs, who share no phenotypical likeness are the same, but Africans who actually do look alike aren't Black lol....
Back to meat:
Here is Whites' theory:
The original Black settlers of Europe TURNED WHITE because they became FARMERS, who ate only farm produce devoid of Vitamin D. And, the lessened solar duration and intensity in the Northern regions REQUIRED a Whitening of the skin for faster absorption of UV radiation and production of Vitamin D. So then, White skin absorbs Sunlight FASTER? Let's see.
1) Modern Whites in Europe are Germanics, Slavs, and Turks. They have nothing to do with ancient ANYBODY in Europe. They are migrants from Central Asia in the CURRENT era. Any encyclopedia will document their entry into Europe.
2) These Whites in Central Asia were Nomads - NOT FARMERS! Their diet was rich in Meats and FISH! Plus they had plenty of Sunshine - in Kazakhstan-south, for eight months of the year, the average UV intensity is level 8 out of a maximum 11. Thus there was no reason to turn White there!
3) Whites did NOT bring agriculture to Europe, Blacks from the South did, and they did NOT turn White.
4) European Whites in the early current era were NOT farmers - Farming is very RECENT to European Whites!.
5) The Whitest of the Whites in Europe - the Germans, inhabited Costal WESTERN Europe and Scotland - regions rich in fish and game animals - ancient Germany and modern Germany are different areas.
The Roman historian Cornelius Tacitus (56-118 A.D.) said this about them: For my own part, I agree with those who think that the tribes of Germany are free from all taint of intermarriages with foreign nations, and that they appear as a distinct, unmixed race, like none but themselves. Hence, too, the same physical peculiarities throughout so vast a population. All have fierce blue eyes, red hair, huge frames, fit only for a sudden exertion. They are less able to bear laborious work. Heat and thirst they cannot in the least endure; to cold and hunger their climate and their soil inure them. Does THAT sound like FARMERS?
During the time of Tacitus, Germans inhabited Caledonia (Scotland), the rest of Britain was inhabited by Blacks, they were the original settlers of Britain, and STILL BLACK! Tacitus describes them thusly: "The DARK complexion of the Silures, their usually curly hair, and the fact that Spain is the opposite shore to them, are an evidence that Iberians of a former date crossed over and occupied these parts. Those who are nearest to the Gauls are also like them, either from the permanent influence of original descent, or, because in countries which run out so far to meet each other, climate has produced similar physical qualities."
Moving along, let's examine some other things huh?
6) A Human being requires ONLY 15 minutes of Sunshine, on face or hands, TWICE a week, for good health! Thus no reason for Black people to turn White.
But for the sake of argument, lets accept this bit of White myth, that Blacks TURNED White because of Vitamin D deficiency - Lets test it. Think of the absurdity of this: Black humans survived in Europe for about 35,000 years, then all of a sudden, their bodies were not getting enough vitamin "D": forcing them to lighten their skin. The problem is: logically, BEFORE there could be any evolutionary movement toward lighter skin - the population would have DIED OUT from Rickets! This supports Albinism, but there were Blacks
Rickets
Rickets is a softening of bones in children due to deficiency or impaired metabolism of vitamin D, phosphorus or calcium, potentially leading to fractures and deformity. The Roman physician Soranus reported deformation of the bones in infants as early as the first and second centuries AD. He attributed these deformities as resulting from the failure of Roman mothers to properly nurture and clean their children, but didn't directly implicate poor diet in the condition. Rickets wasn't defined as a specific medical condition until 1645, when an Englishman named Daniel Whistler gave the earliest known description of the disease while still studying medicine.
Rickets was very common in industrialized cities around the turn of the twentieth century, both in the United States and in Europe. It was most common among the poorer, less privileged people and often affected infants. Still, there was not a complete understanding that the condition was caused by poor nutrition, although it was considered a potential cause, along with lack of fresh air and sunshine. In the late nineteenth century, strides had been made toward finding a cure for rickets. At the London Zoo in 1889, lion cubs were fed a diet of boneless lean meat. When some cubs developed florid rickets, they were treated by the addition of cod liver oil and crushed bones to their meals, and made a full recovery.
What causes rickets? Regardless of the type of rickets, the cause is always either due to a deficiency of vitamin D, calcium, or phosphate. Three common causes of rickets include nutritional rickets, hypophosphatemic rickets, and renal rickets.
So to carry the absurdity to its logical conclusion: supposing that these Black people turning White, survived thousands of years of Rickets - in a non-deformed state, and then appeared as modern Whites. Then why would MODERN Whites STILL get Rickets? Wasn't the whole point of evolution to develop White skin, just for the "SUPPOSED" reason that Sunlight could be quickly and efficiently absorbed by the White skin, just to AVOID such things? You would have built up the resistance to sustain Rickets. Albinism is the stabilized norm for White skin, but there are variations of Albinism and not all lost total pigmentation.
